Never been here before….
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Never been here before….
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meanwhile…
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Men and women may in the end be forced to admit: “I made a fool of myself,” and still be fairly happy. But no one can possibly be satisfied, and therefore no one can in my sense be happy, who feels that in some paramount affair he has failed to take up the challenge of life. For a voice within him, which none else can hear, but which he cannot choke, will constantly be murmuring:
“You lacked courage. You hadn’t the pluck. You ran away.”And it is happier to be unhappy in the ordinary sense all one’s life than to have to listen at the end to that dreadful interior verdict.
~ Brett McKay from, “Running Away From Life” Essay by Arnold Bennett | The Art of Manliness
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Instagram to avoid burning myself. NY pizza YASSSSSSSS!
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Long-term consistency trumps short term intensity.
~ Bruce Lee
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The diet-heart hypothesis is the idea that saturated fat, and in some versions cholesterol, raises blood cholesterol and contributes to the risk of having a heart attack. To test this hypothesis, scientists have been studying the relationship between saturated fat consumption and heart attack risk for more than half a century. What have these studies found?
~ Stephan Guyenet from, The Dirty Little Secret of the Diet-Heart Hypothesis
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Life Return On Exercise Invested – LROEI
Exercise has been show to correlate (caution, https://en.wikipedia.org/wiki/Correlation_does_not_imply_causation) with longevity; The people who exercise more are also the people who live longer:
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I had never thought about exercise in quite this way: If I’m going to exercise, and if the exact details of the exercise aren’t so important, WHICH exercise would I prefer to do?
Would I rather spend time indoors or outdoors?
…in a gym, or at a playground?
…alone, or with my friends?
…in a familiar place, or some place new?
There are many types of exercise. The way I’m pursuing exercise through Parkour ALSO happens to generate a greater LROEI.
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Body fat produces a hormone called leptin, which signals to the brain and other organs to decrease appetite, increase the metabolic rate and increase physical activity. More fat means more leptin, which then causes the extra fat to be burned. The little glitch is that some people become resistant to leptin, so that their brain doesn’t hear the fat tissue screaming that it’s already full. Leptin resistance nearly always accompanies obesity, because it’s a precondition of significant fat gain.
~ Stephan Guyenet from, The Body Fat Setpoint, Part II: Mechanisms of Fat Gain
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This part of his series is short and non-technical. But his whole series is, probably, the greatest explanation of why one gets fat which I have ever read.
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Went for a little hike last weekend with a group. Top-to-bottom: Bear Mountain bridge over the Hudson, View from the top, and a peak of NYC in the far distance. Stellar weather.
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My position, and that of a growing number of researchers, is that there’s more to obesity than calorie balance, and that body weight and fat mass is regulated by a complex system that involves an array of hormones and feedback mechanisms.
~ John Briffa from, «http://www.drbriffa.com/2011/03/10/is-obesity-all-in-the-mind/»
In recent months I’ve become interested in the potential role of a hormone by the name of leptin in obesity.
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It’s not “in the mind” in the sense that you can imagine your way to weight loss. But there’s definitely MORE to weight loss than the balance of calories. My experience is that the best way to lose weight is to simply reduce my calorie intakes slightly — no need to count everything and aim for a computed target intake. At the same time, include some strength training (a ton is not required, but walking/cardio is NOT what worked for me.)
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